2 lectures / chairs 1 badge
Premium

Dr Kristina DAVIDOVIC

Plastic surgeon
Serbia

For the last ten years I have been engaged in aesthetic and antiaging medicine. I am teaching assistant at University of Belgrade, Serbia; founder of national aesthetic society Sesiam and founder of private clinic Rea Medika. I am educator and speaker for many brands: KOL for Skintech, International trainer for Perfectha, Ellanse and SilhouetteSoft, national and regional trainer for Teosyal. 

In the last several years I gave many lectures worldwide related to nonsurgigal face rejuvanation and remodeling, complications, combined treatments, and social media influence, since our social network engage more than 70K real and active followers. In the last years our private practice where I am the leading doctor become one of the most famous and the most popular clinic at entire region, allowing us to have more than 5000 patients per year and more than 10000 injections and other procedures per year. Being winner of many national and international scholarships allowed me to exchange my knowledge and expertise in order to outstand my expertise and transfer all the data to my students. 

IMCAS is one of the most important scientific and educational platform, since it gave me differen perspective on teaching and academical aspects. It gathers at one place the best scientists, injectors and brands at one place aiming improving our skills and knoledge. 

read more
add item add Kristina DAVIDOVIC

Kristina DAVIDOVIC's publications (2)

Clinical, cognitive, and behavioural correlates of white matter damage in progressive supranuclear palsy.

May, 2014

White matter (WM) tract alterations were assessed in patients with progressive supranuclear palsy (PSP) relative to healthy controls and patients with idiopathic Parkinson's disease (PD) to explore the relationship of WM tract damage with clinical disease severity, performance on cognitive tests, and apathy. 37 PSP patients, 41 PD patients, and 34 healthy controls underwent an MRI scan and clinical testing to evaluate physical disability, cognitive impairment, and apathy. In PSP, the contribution of WM tract damage to global disease severity and cognitive and behavioural disturbances was assessed using Random Forest analysis. Relative to controls, PSP patients showed diffusion tensor (DT) MRI abnormalities of the corpus callosum, superior cerebellar peduncle (SCP), cingulum and uncinate fasciculus bilaterally, and right inferior longitudinal fasciculus. Corpus callosum and SCP DT MRI measures distinguished PSP from PD patients with high accuracy (area under the curve ranging from 0.89 to 0.72). In PSP, DT MRI metrics of the corpus callosum and superior cerebellar peduncles were the best predictors of global disease severity scale scores. DT MRI metrics of the corpus callosum, right superior longitudinal and inferior longitudinal fasciculus, and left uncinate were the best predictors of executive dysfunction. In PSP, apathy severity was related to the damage to the corpus callosum, right superior longitudinal, and uncinate fasciculi. In conclusion, WM tract damage contributes to the motor, cognitive, and behavioural deficits in PSP. DT MRI offers markers for PSP diagnosis, assessment, and monitoring. read more

Journal of neurology

White matter abnormalities in Parkinson's disease patients with glucocerebrosidase gene mutations.

Jun, 2013

Glucocerebrosidase gene mutations represent a genetic risk factor for the development of Parkinson's disease. This study investigated brain alterations in Parkinson's disease patients carrying heterozygous glucocerebrosidase gene mutations using structural and diffusion tensor magnetic resonance imaging. Among 360 Parkinson's disease patients screened for glucocerebrosidase gene mutations, 19 heterozygous mutation carriers (5.3%) were identified. Of these, 15 patients underwent a neuropsychological evaluation and a magnetic resonance imaging scan. Sixteen age- and sex-matched healthy controls and 14 idiopathic Parkinson's disease patients without glucocerebrosidase gene mutations were also studied. Tract-based spatial statistics was used to perform a white matter voxel-wise analysis of diffusion tensor magnetic resonance imaging metrics. Mean fractional anisotropy values were obtained from white matter tracts of interest. Voxel-based morphometry was used to assess gray-matter atrophy. Cognitive deficits were found in 9 mutation carrier patients (60%). Compared with controls, Parkinson's disease patients carrying glucocerebrosidase gene mutations showed decreased fractional anisotropy in the olfactory tracts, corpus callosum, and anterior limb of the internal capsule bilaterally, as well as in the right anterior external capsule, and left cingulum, parahippocampal tract, parietal portion of the superior longitudinal fasciculus, and occipital white matter. Mutation carrier patients also had decreased fractional anisotropy of the majority of white matter tracts compared with Parkinson's disease patients with no mutations. No white matter abnormalities were found in Parkinson's disease patients without glucocerebrosidase gene mutations. No gray matter difference was found between patients and controls. In Parkinson's disease patients, verbal fluency scores correlated with white matter abnormalities. Parkinson's disease patients carrying glucocerebrosidase gene mutations experience a distributed pattern of white matter abnormalities involving the interhemispheric, frontal corticocortical, and parahippocampal tracts. White matter pathology in these patients may have an impact on the clinical manifestations of the disease, including cognitive impairment. read more

Movement disorders : official journal of the Movement Disorder Society